Prenatal immune activation potentiates endocannabinoid-related plasticity of inhibitory synapses in the hippocampus of adolescent rat offspring
There is strong evidence that immune activation from prenatal infection increases the risk for offspring to develop schizophrenia. The endocannabinoid (eCB) system has been implicated in the pathophysiology of schizophrenia while models of cortical dysfunction postulate an imbalance between neuronal...
|Published in:||European neuropsychopharmacology : ENP : the journal of the European College of Neuropsychopharmacology, Vol. 28, No. 12 (2018), p. 1405-1417|
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European neuropsychopharmacology : ENP : the journal of the European College of Neuropsychopharmacology, Vol. 28, No. 12 (2018), p. 1405-1417